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Direct myocardial cell harm might happen by disruption of mitochondria and induction of intrafibrillar collagen dysplasia (Melchert and Welder 1995; Sullivan et al 1998). Cellular damage results in fibrosis, which can be a potential nidus for ventricular arrhythmias. Patients generally develop left ventricular hypertrophy, which may be associated to hypertension, one other adverse effect of AAS. The left anterior artery demonstrates a 95% stenosis after the first septal department. Scientific and technological companies and analysis and design relating thereto; industrial analysis and analysis services; design and growth of laptop hardware and software.
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